Just Hubris?

www.morphostasis.org.uk

Just hubris?

...... if you like.

But how else do I make people listen and understand without ramping up the provocation?

And this might get you to realise what I believe (it's in the same vein as morphostasis and the zygote derived colony):

"It is a matter of perfect indifference where a thing originated; the only question is: is it true in and for itself?" G. W. F. Hegel, 1770-1831.

No one person can create any concept out of a vacuum. Whatever proves to be the "correct" conception will be a 99.99999% communal intellectual activity that starts with us learning to speak, is expanded in our higher education and culminates in the communal application of that education and our aggregated knowledge. A single neurone doesn’t make a mind any more than a single brain commands an intellect. Intellect is far more an emergent property of the communal than the individual self. I hope no one feels that I am in any way or at any time thinking of claiming sole "animal" rights for any of these concepts. History, at least, has always solved that one. There are always a number of minds thinking along the "realisation" lines. We can trust historians to squabble over the question of credits - like jackals over a carcass. Anyway, does that matter at all when understanding is at stake?

I suspect there is some annoyance that I don’t reel off all the relevant credits to those who have evolved concepts of importance in immunology. I’m not a historian of immunology – to me it’s a hobby/obsession. Medical Hypotheses asks authors to keep the references to a frugal minimum.

I'd like to add one thought of my own; that merit should be attributed to the conception rather than to the conceiver. Should the conception I have proposed be dominantly right then the important object is to get it tidied up and weed out the rubbish that I have undoubtedly introduced.

It is worth asking yourself this set of questions:

	Do phagocytes have anything to do with morphostasis? (ie, "tissue homeostasis")
	Do gap junctions have anything to do with morphostasis?
	Is gap junction activity altered when a healthy cell becomes sick?
	Do gap junctions have a function in alerting adjacent cells of a problem?
	Do gap junctions help spread a wave of suicide to adjacent cells under certain circumstances (like the plant hypersensitivity response).
	Are gap junctions important in macrophage activity?
	Is it possible to look at everything we have considered to be self/non-self (or pathogen/non-pathogen) discrimination and restructure it under a morphostasis paradigm? (This is rather like holding back the tide of entropy – Zlatko Dembic’s approach).

This page was last updated on 04/10/04